Diversity in America Essay Example for Free

Diversity in America Essay In an ideal world, humanity would understand that all mankind is created equally; that the underlying truth of each of us is goodness, and that through awareness, conscious choice and the willingness to create positive change, we could live in a world where diversity is celebrated. We would leave behind the substantial racist and oppressing patterns that exits in this world, specifically in the United States of America. It is said that the U. S. is a melting pot of cultures, and that we are a country of immigrants existing together as a new culture, living under the values of a democracy based on freedom, liberty and the pursuit of happiness. Yet this policy is completely disregarding the fact that as immigrants, we brutally committed genocide to the Native Americans. The dominant race in the U. S. A. is made up of white Euro-centric people, and looking at the patterns that exist in this culture is important to examine the history, the ways in which racism is alive and how and who is affected. We all must look at how, as an individual and within a community, we can work towards positive change, healing and understanding. As a society, we have committed and perpetuated the oppression of different cultures specifically the Native Americans, the Native Africans and the many immigrants from different countries. In the early history of the U. S. government, it is clear that there was a systematic method that aimed to remove the Native Americans from the land that was desired by the colonists, with the malicious intention to commit genocide. The first example of the patterns of racism that were established is seen in the fabrication of stereotypes onto the Native Americans. It was said that the Natives were â€Å"barbarians† and that they would rape and murder women and children and that they â€Å"served the devil† (Tataki, 1993, p. 41). The whites held the belief that the Natives were occupying land that the colonists felt entitled to. â€Å"White people also justified the genocide by saying that Native Americans died from diseases they were biologically unable to resist† (Kivel, 2002, p.126). It is a known fact that smallpox were given to the Natives as a way to kill them. Multiple examples exist throughout the history of the whites murdering, raping and unjustly exploiting almost every aspect of the Native’s culture. After committing such horrendous violations we are left with the inability to change all that has occurred and a great sadness that produces guilt, blame and anger that often stagnates a healing process and increases denial and avoidance. The Native American population has almost completely been destroyed. â€Å"At the time Columbus arrived in the West Indies there were approximately fifteen million indigenous people†¦ today†¦ the population of native Americans in the United States is around three million according to U. S. government census figures† (Kivel, 2002, p. 124) and the remaining Natives in America are mostly confined to reservations. This small fraction of designated land is no longer their original sacred land but it is being raped for natural resources. White settlers not only committed genocide but they also enslaved the Native Americans. This pattern of entitlement and abuse was continued with the legal capturing and enslavement of people of African decent with as much violence and oppression. The history of slavery in the United States that occurred through 1619 to 1865 began soon after the English colonists first settled in Virginia and lasted until the passage of the thirteenth Amendment to the U. S. Constitution. â€Å"Over the next twenty five years Virginia passed a series of laws that legalized slavery, producing a radically subordinate and stigmatized class below that of all whites† (Kivel, 2002, p.130). Although technically slavery was abolished in1865, a linage of abuse and inhumane treatment was installed and has been carried into this day and age providing a challenge to accept and comprehend the past. In an attempt in understanding black oppression, there are aspects that demonstrate this injustice. They are institutional racism, racist knowledge and power relations that are played out in our culture and in no way have anything to do biology. Individuals and societies have created and used race as a means to oppress and overpower other groups of people. Racial oppression is when a group of people dominates another for their own benefit disregarding justice and respect through the use of violence and defining and discriminating racial differences. This dominant group receives various benefits although in the larger picture all sides loose for the continuation of a pattern of pain and injustice is insured through these actions. African-Americans are a case of this racial oppression. They were turned into slaves because of the color of their skin. It is shocking that it did not start this way and that through the power of the U. S.government slavery laws were passed that enabled the white masters to turn the blacks into slaves. This is an example of the institutional racism used to enslave the blacks. Because of this occurrence, we, as a society, must break down the residual stereotypes that have instilled fear, pain and disconnection between the races, and to change the model that exists even at this point in time. Another example of racism in the U. S. is seen in the treatment of immigrants. This subject is personal, for on my father’s side of my family I am part of the first generation born in American. My father’s parents immigrated to the U. S. , to escape the holocaust and I am sure shared the dreams of the majority of different immigrants who traveled to the â€Å"land of opportunity,† escaping places of war and economic devastation to begin and pursue a new and better life. Through the duration of attending a class studying the diversity in America I have gained painful yet poignant knowledge of the racism that is still perpetrated upon immigrants, specifically on Jewish people. I have recently learned that groups of neo-Nazis congregate and commit acts of violence against Jewish people and immigrating races. This is terrifying to me and feels unacceptable while we live under a constitution that allows personal expression but does not permit such distinct racist and violent behavior. I am grateful and saddened that because I was raised in a protected and privileged community I have rarely experienced oppression and hateful discrimination when it so readily exists in our culture. In the past few months I find myself cycling through heartbreak, anger and disbelief of the injustice that has and still occurs, and then to a yearning for healing and equality for all. I remain in a space of wonderment, questioning the fact that although laws have been installed to prevent the acts of racism, fear, ignorance and violence is bubbling hot under the surface of our society, and we are a long way from a complete shift in humanity that I crave. I do believe there is hope. I believe that in gaining the truth of the past and diminishing ignorance of the harm that was and still is being done we open a door that may aid in the battles that are still being fought. Although the brutality of racism is alive, the potential to fight for the rights of all the people who live upon this American soil is possible, but the truth of the history and the attainment of awareness must be brought to fruition. Reference List Kivel, Paul, (2002). Uprooting racism: How White People Can Work For Racial Justice. Gabriola Island, BC VOR 1X0, Canada: New Society Publishers. Takaki, Ronald, (1993). A Different Mirror: A History of Multicultural America. New York, NY: Time Warner Book Group.

Aetiology and Pathophysiology of Heart Failure

Aetiology and Pathophysiology of Heart Failure Rachel Corston-Jackson Heart Failure The heart is a diverse organ and the diseases associated with it are caused by many different organs when they become dysfunctional or diseased. It is for this reason that heart failure is more diverse than just the stopping or failure of the heart as there are many types and causes of heart failure. Therefore, this essay will discuss the subject heart failure in the human body by addressing its aetiology and pathophysiology, the signs and symptoms associated with it, as well as the risk factors and causal agents linked to heart failure, and lastly, the relevant tests and treatment options available to heart failure patients to improve their quality of life. Aetiology: Heart failure, sometimes called congestive heart failure, refers to when the heart isnt pumping blood as well as it should (American Heart Association, 2014). It is a chronic disease characterised by the failure of the heart as a pump and is the result of any structural or functional cardiac disorders (Butler, 2012). This doesnt mean that the heart stops beating, however, the American Heart Association (2014) warns that heart failure can get worse if its not treated. The heart does not stop altogether but keeps working, despite the fact that the demands of blood and oxygen of the heart and body far exceed supply (Butler, 2012). This is caused a variety of disorders such as coronary artery disease, heart attacks, cardiomyopathy, and congenital birth defects (Butler, 2012). Coronary artery disease (CAD), as stated by Butler (2012) is a disease characterised by a narrowing or blockage of the arteries which restricts blood and oxygen supply to the heart, resulting in a reduced preload in the ventricles and reduced ventricular stretch. When low ventricular stretch occurs it results in a decreased force of cardiac contraction and a low stroke volume. The effects of low stroke volume include low blood pressure and can lead to the body’s organs and tissues becoming deprived of adequate oxygen and nutrients which may cause the body to go into a state of shock. The second cardiovascular disorder which can cause heart failure is a heart attack. A heart attack according to the Heart Foundation (2015) occurs when ‘a coronary artery becomes suddenly blocked, stopping the flow of blood to the heart muscle’. Damage to the heart muscle occurs during a heart attack, and becomes scarred, the damaged area does not function properly, resulting in a reduced cardiac output and low blood pressure. When this happens the heart compensates by undergoing a remodelling process where it changes in size, shape or structure, and according to Butler (2012) the remodelling is more likely to occur in the left ventricle as it has a thicker muscle mass. The third form of heart failure mentioned above is cardiomyopathy, which is caused by infections, alcohol abuse, or pregnancy and is characterised by damage to the heart muscle. The type of cardiomyopathy which presents during pregnancy is called peripartum cardiomyopathy (Demir, Tufenk, Karakaya, Akilli, Kanadas, 2013). It is a form of dilated cardiomyopathy and involves systolic dysfunction of the heart. Onset is usually around the last month of pregnancy and five months postpartum, hence the name. One common symptom of peripartum cardiomyopathy is sinus tachycardia which according to Demir et al. (2013) can be treated with a drug called Ivabradine. This brings me to the next section which is the pathophysiology of heart failure. Pathophysiology: Heart failure is a complex problem and is characterised by many signs and symptoms. Symptoms include; shortness of breath, orthopnoea, paroxysmal nocturnal dyspnoea, fatigue, reduced ability to exercise, peripheral oedema, loss of appetite and more (Nicholson, 2014). Signs include; Tachycardia, tachypnoea, an abnormal pulse, and displaced apex beat, third heart sounds, a raised jugular venous pressure, lung crepitation, weight changes, hepatomegaly and more (Nicholson, 2014). The common symptoms which will be focused on here include shortness of breath (S.O.B), paroxysmal nocturnal dyspnoea, fatigue, and a lack of appetite (Butler, 2012). The American Heart Association (2014) states that S.O.B during activity, at rest, or while sleeping, can have a sudden onset, often causing the patient to wake. They describe S.O.B as being caused by the blood when it backs up in the pulmonary veins because the heart cant keep up with the supply. The result is stated to be that the kidneys develop a reduced capacity to dispose of sodium and water, and that excess fluid leaks into the lungs causing a pulmonary oedema and around the lungs causing pleural effusion. The clinical sign which indicates that this has occurred, aside from S.O.B, is the presence of lung crepitations (Butler, 2012). A lung crepitation, according to Butler, is a crackling sound produced during inhalation and exhalation due to fluid accumulation inside the lungs and predominantly occurs in the lower lung fields. The American Heart Association states that peripheral oedemas can also occur as a result of the venous blood backing up and is characterised by swelling in the ankles, feet and abdomen resulting in ill-fitting shoes and weight gain due to the excess fluid accumulation. The build-up of fluid which causes S.O.B also leads to paroxysmal nocturnal dyspnoea (PND) (Butler, 2012; Nicholson, 2014). PND usually occurs at night, according to the American Heart Association, and is characterised by sudden awakening from sleep after only a few hours, with a feeling of breathlessness, suffocation and severe anxiety. On chest auscultation, the bronchospasm associated with a heart failure exacerbation can be difficult to distinguish from an acute asthma exacerbation (Dumitru Baker, 2014). Other symptoms of heart failure included fatigue. Fatigue and weakness according to Dumitru Baker (2014) are often accompanied by a feeling of leaden limbs and are generally related to poor perfusion of the skeletal muscles in patients with a lowered cardiac output. Essentially the heart produces a decreased volume of blood and cannot meet the needs of body tissues so the body diverts blood away from less vital organs, particularly muscles in the limbs, and sends it to the heart and brain (American Heart Association, 2014). The American Heart Association (2014) and Nicholson (2014) both link this diversion of blood to vital organs to the loss of appetite and feelings of nausea often experienced by people with heart failure due to the fact that the digestive system receives less blood and cannot function correctly. The common signs that will be discussed here are hepatomegaly, third heart sounds, tachycardia and a displaced apex beat. Hepatomegaly, an enlargement of the liver which can occur with right heart failure (Nicholson, 2014), and is caused by the blood backing up from the heart into the inferior vena cava, such congestion increases pressure in the inferior vena cava and other veins that carry blood to it, including the hepatic veins (Orfanidis, 2013).Once this occurs the pressure may build to a point where the liver becomes engorged with blood and malfunctions. The common symptoms of hepatomegaly are nausea, abdominal pain or fullness, swelling of the feet and legs, and shortness of breath, all of which are also seen in heart failure (Butler, 2012). The third heart sound (S3) is a low-pitched sound that occurs when the ventricles fill rapidly and is one of the more specific signs of heart failure and auscultating to determine the presence of it can help healthcare professionals to diagnose heart failure (Santhosh, 2009). S3 is commonly present in conjunction with tachycardia, which is a high resting heart rate and is common as a haemodynamic compensatory response (Nicholson, 2014). The final sign of heart failure listed above is a displaced apex beat. A displaced apex beat means ‘the point of maximal impulse on the precordium can be displaced down and to the left laterally and commonly occurs when the heart is dilated (Nicholson, 2014, p. 33)’. Each sign and symptom of heart failure is linked to a causal agent such as a previous history of Myocardial Infarctions (MI). Risk factors/causal agents: The term ‘heart failure’ includes many conditions and disease, thus it has many causal agents including; Family history, narrowed arteries, high blood pressure, coronary artery disease, myocardial infarctions, valve disorders, peripartum, ischemic heart disease, diabetes, obesity, kidney disease, hypothyroidism, toxins (alcohol), and infection (Butler, 2012; Nicholson, 2014). The causal agents which will be discussed here are narrowed arteries, high blood pressure, obesity, diabetes and infection. Family history is an important indicator of increased risk in relation to heart failure, it is however not to be considered on its own but in relation to other risk factors such as narrowed arteries (Goldberg, 2014). Narrowed arteries can refer to vasoconstriction or atherosclerosis. Atherosclerosis is a plaque formation is medium or large sized arteries in response to damage of the tunica intima (National Health Council, 2014). These plaque formations cause increased resistance to laminar blood flow resulting in turbulent flow and high blood pressure (Foss Farine, 2013). Blood pressure alone is characterised as the force exerted on blood vessel walls by a volume of blood as it passes through (Heart Foundation, Blood Pressure, 2010). High blood pressure on the other hand is known as hypertension, and is defined by the Heart Foundation (2010) as chronically elevated blood pressure resulting in stain on the heart and blood vessels. Hypertension is visually manifested by jugular venous distention on the right side of the neck (Nicholson, 2014). Foss Farine (2013) state that the elevated blood pressure is detected in the blood vessels by baroreceptors located in the carotid sinus and aortic arch. The baroreceptors are said to then stimulate vasomotor nerves to increase the diameter of the blood vessels to increase blood flow and reduce blood pressure. Another cause of high blood pressure is obesity. This is due to the fact that overweight or obese people have a greater the volume of tissue and fat that requires a constant blood supply, this results in an increase in blood vessel length (Foss Farine, 2013). Foss Farine (2013) state that the longer the blood vessels become the more distance the blood will have to travel which increases the resistance, the body will compensate for this by increasing the blood pressure throughout the body by increasing the stroke volume of the heart. This increase in blood pressure is to ensure both adequate blood supply to all blood vessels and adequate venous return. If the high weight threshold is maintained then high blood pressure will continue, causing hypertension and heart strain (Heart Foundation, Blood Pressure, 2010). A second effect that obesity can have on the heart is diabetes mellitus. Diabetes is a disorder of the metabolism where the glucose produced from the breakdown of food is no effectively absorbed into the cells for fuel. Diabetes is characterised by an inadequate production of the hormone called insulin which is produced in the pancreas and must be present to allow glucose to enter the cells (Goldberg, 2014). When insulin production is low the glucose remains in the blood and has many effects, such as increasing the susceptibility to infection (Foss Farine, 2013). The long term effects of low insulin and high glucose levels in the blood include atherosclerosis, which is an increase in deposits of fatty materials on the insides of the blood vessel walls (Goldberg, 2014). These deposits affect blood flow by reducing the diameter of the blood vessels and raising blood pressure, increasing the chance of clogging and hardening of blood vessels (Goldberg, 2014). Relevant tests: There are many tests which can help determine if a patient has heart failure or is at risk, such as; checking blood pressure, chest x-rays, blood tests, 12-lead electrocardiogram and respiratory function tests. Checking blood pressure regularly is part of a standard visit to the doctor or nurse, because it helps to establish a pattern of high, normal or low blood pressure. Long term high blood pressure has been linked to heart strain, and in conjunction with other health issues such as atherosclerosis can result in heart failure. (Heart Foundation, Blood Pressure, 2010). Another test which can contribute to the diagnosis of heart failure is a chest x-ray. Chest x-rays may be performed to look for signs of a pulmonary oedema which can cause symptoms such as S.O.B and paroxysmal nocturnal dyspnoea (Butler, 2012). A pulmonary oedema is commonly caused by a disrupted flow of blood to and from the heart. Respiratory function tests are also performed, to exclude respiratory causes for dyspnoea, such as asthma and chronic obstructive pulmonary disease (COPD) (Butler, 2012). Another direct test of the heart is via a 12-lead electrocardiogram (ECG), the results of an ECG may show evidence of left or right ventricular hypertrophy, CHD, or arrhythmias commonly associated with heart failure such as atrial fibrillation (Butler, 2012). If no abnormality is present then the patient is unlikely to have heart failure (Nicholson, 2014). Further tests for signs of heart failure include blood tests for glucose (Butler, 2012; Nicholson, 2014). Checking glucose levels can be performed at home and is a vital component in the management of diabetes because if the blood glucose level remains high it can lead to atherosclerosis, and later, heart failure. High glucose levels are managed by first testing the blood then administering the appropriate amount of insulin to help absorption of the glucose into the cells and thus lower the blood glucose levels. Treatment strategy: Education on self-management strategies is a vital aspect of patient empowerment and care both at hospital and in their own home (Cockayne, Pattenden, Worthy, Richardson, lewin, 2014). Heart failure patients require education how they can manage their symptoms and to ensure they can recognise the warning sign associated with acute situations (Nicholson, 2014). Patient education commonly includes information on how to maintain good control of comorbid conditions such as diabetes. Control of diabetes at home begins with the monitoring blood glucose levels and commonly results in injecting oneself with insulin (Goldberg, 2014). Cockayne et al. (2014) state that ensuring patients understand the importance of adhering to the medication regime designed for their optimal health is a vital part of patient education. Another important aspect of patient education and self-management is providing an explanation on how to improve health habits and adopt a healthier lifestyle (Nicholson, 2014). The most commonly required lifestyle change to improve health outlooks for patients is to advise that they limit any consumption of alcohol and tobacco smoke as both produce detrimental effects on the heart, such as, causing pulmonary blockages and narrowing of airways, which will affect the oxygen supply to the blood, tissues and organs (Nicholson, 2014). Other important lifestyle changes according to Nicholson (2014) and Butler (2012) include advice on losing weight if the patient is obese, because obesity results in long, narrow blood vessels and high blood pressure which increases the patient’s risk of heart failure. The management of weight for patients with heart failure includes recommendations for specific dietary changes such as a low sodium intake or a low refined sugar intake (Butler, 2012). It may also include guidelines for increased daily exercise which is shown to have positive effects on heart failure symptoms according to Nicholson. Butler (2012) states that for women there is another important lifestyle factor to consider in the self-management of heart failure symptoms, the use of contraception. Contraception is important for women who experience heart failure and its symptoms because if a woman with heart failure were to become pregnant it would increase her risk of heart failure and morbidity during pregnancy and birth. The use of pharmacology in the management and treatment of heart failure symptoms is multifaceted. The use of angiotensin-converting enzyme (ACE) inhibitors is said to be one of the most valuable drug therapies in heart failure according to Butler and is intended to decrease the effects of compensatory mechanisms which are maladaptive so as to improve heart failure symptoms and increase the rate of survival, particularly when taken in conjunction with beta-blockers. Beta-blockers work by reducing heart rate and the myocardial oxygen demand (Nicholson, 2014). The use of ACE inhibitors means that it is important to closely monitor the blood chemistry of the patient, and that the side effects are commonly limited to a dry, persistent cough (Butler, 2012). If the patient cannot tolerate ACE inhibitors due to the dry, persistent cough then the use of angiotensin-II receptor blockers (ARBs) will be considered as this drug has similar properties to the ACE inhibitor and will also require th e monitoring of blood chemistry. (Butler, 2012). Other pharmacological treatments include the use of vasodilators and diuretics. Vasodilators are used to improve cardiac output and often used in cases of heart failure where the patient cannot tolerate ACE inhibitors or ARBs (Butler, 2012). Diuretics are used to increase fluid loss in order to reduce the size and occurrence of peripheral oedemas and pulmonary oedemas, resulting in a reduced level of breathlessness (Nicholson, 2014). The last treatment option to be discussed here is the use of device therapy. Device therapy as stated by Butler (2012) refers to an implantable cardiac defibrillator (ICD) and a biventricular pacemaker. Biventricular pacemakers are implanted to restore ventricular synchrony and reduce symptoms in the event ventricular failure. ICDs on the other hand addresses the problem of cardiac death by delivering an electric shock to the heart to restore normal rhythm and function. In conclusion, heart failure is a complex and multifaceted health problem which encompasses many heart problems like coronary artery disease, heart attacks and cardiomyopathy. Because heart failure is such a large problem it has many symptoms including S.O.B, fatigue and oedemas, and also many signs like tachycardia and lung crepitation. Each sign and symptom of heart failure is associated with a causal agent or risk factor such as hypertension in relation to obesity and diabetes. Heart failure cannot be cured, however the symptoms can be managed through patient education and self-management, pharmacology, and as a last resort, device therapy. 1 | Page

UK Law on Voluntary Manslaughter

UK Law on Voluntary Manslaughter Jocelyn Ayres Diminished responsibility.This is one of the three special defences which exist for the defence of murder. It is contained in the Homicide Act of 1957 and is modified by the Coroners and Justice Act 2009. When the defence of diminished responsibility is pleaded successfully it can reduce a murder conviction to manslaughter. To use diminished responsibility the defendant must be able to demonstrate one of the following: An abnormality of mental functioning caused by a recognised medical condition. The question of whether the defendant is suffering from the abnormality of a mental functioning is decided by the jury after hearing medial evidence.   The jury dont have to follow the medical evidence. Some examples of what counts as an abnormality of the mind include Jealousy, Battered woman syndrome, pre-menstrual tension, Epilepsy and chronic depression. An example of this is R v Byrne. The appellant murdered a young girl staying in a hostel, and mutilated the body. He did so as he was suffering from irresistible impulses which he was unable to control. Abnormality of mind was wide enough to cover the minds activities including the ability to exercise will power. He was allowed the defence to reduce the charge to manslaughter. The abnormality must provide an explanation for defendants act or omission in being party to the killings.   This follows from the old law which required the abnormality to be caused by an arrested or retarded development of the mind or induced by injury or disease. Alcohol and drugs are not considered unless the abnormality is alcoholism or drug addiction. Something like binge drinking doesnt allow the defendant to use diminished responsibility. Case law for this could come from R v Wood. The appellant was an alcoholic who had been sleeping rough. He was friends with a group called the breakfast club and had been drinking heavily with them for the past two days before the attack. He was invited to spend the night at the deceaseds house. During the night, he woke up and found the deceased trying to perform oral sex on him, he attacked him with a meat cleaver and hammer killing him. The judge said that a mans act is involuntary if, and only if, it is it very difficult to do otherwis e. The appellant appealed this and the conviction for murder was quashed. Which substantially impaired his/her mental ability. The defendant must show that the abnormality of the mind must have substantially impaired their mental ability to either understand the nature of their conduct, form a rational judgement or to exercise self-control. This is something for the jury to decide after hearing the medical evidence. A case example for this diminished responsibility is R v Campbell. The appellant killed a female hitch hiker he had picked up when she refused his sexual advances. She wanted to go from Oxford from London. He pulled up at a remote spot and made a pass at her. She hit him in the eye and he punched her in the throat. She began gurgling and blood came from her mouth. Realising the force he must have hit her he panicked and strangled her. He killed her by hitting her with a hockey stick. The appellant had frontal lobe damage and epilepsy. He appealed for diminished responsibility with medical opinion that the effect of his epilepsy and lobe damage affected his judgement, control of emotions and impulses and forward planning. His conviction for murder was ended and he went to retrial. Provocation.Provocation is part of the special defences for murder, like diminished responsibility. The requirements for the defence of provocation are: There must be evidence of provocation. This requires there to be evidence that the person charged was provoked by things done or said. There is no requirement that the provocative act was deliberate or aimed at the victim. Even something like the constant crying of a child can be used as evidence. Without the evidence the judge cannot put the issue of provocation to the jury. The jury can consider actions that happen over a period. The defendant is allowed the defence if they induced the provocation. A case example for this is R v Johnson. The appellant was at a night club. A woman called him a white nigger. The appellant was white but had taken to adopting a west Indian accent. He took exception to the comment and made threats to her. A male friend of the woman intervened and poured a drink over the appellant. A fight happened and the appellant stabbed the man and he died. He argued that he was acting in self-defence. The judge directed the jury on self-defence, which they rejected, but did not direct them on provocation. He was convicted for murder which he appealed, and it was swapped for manslaughter because of the provocation. Loss of control. This requires the accused to be provoked into losing their self-control. The definition of provocation came from R v Duffy, and is some act, or series of acts which would cause in any reasonable person and causes in the accused, and sudden and temporary loss of self-control, rendering the accused so subject to passion as to make him or her for the moment not a master of his or her mind. If there is any evidence of planning this will demonstrate no sudden and temporary loss of control. The loss of control need not be complete to negate murderous intent. Case law relevant to this is R v Richens. At the age of 17 the appellant killed a man who had raped his girlfriend. The deceased had taunted him about the rape saying that his girlfriend wanted to have sex and that she had enjoyed it. At which point the appellant stabbed him. The defendant raised the defence of provocation at the trial and the judge directed the jury on loss of control, saying that it doesnt apply to l osing that kind of control, and that it is a complete loss of control, to the extent that you dont know what youre doing. He appealed against the jurys conviction of murder. The provocation must be such as to make a reasonable man do as the defendant did. This is a question for the jury who must balance the gravity of the provocative act against the actions expected of a reasonable man. This is problematic when courts try to interpret and apply the section and it gets appealed regularly. Originally it was objective and no account could be taken of characteristics of the defendant in assessing this. However, it was accepted that particular characteristics could be taken into account, and this lead to some uncertainty. The characteristics must be sufficiently permanent. Some case law for this part of provocation in R v Ahluwalia. The appellant poured petrol and caustic soda on her sleeping husband and then set him on fire. He died six days later from the injuries. The couple had an arranged marriage and the husband had been violent and abusive throughout the marriage, he was also having an affair. On the night of the killing he had threatened to hit her wi th an iron and told her he would beat her the next day if she didnt give him money. At the trial, she admitted to killing him, but used the defence of provocation. She was however, convicted of murder. She appealed this decision and raised diminished responsibility. Diminished Responsibility Case Mary and Peter Abnormality of the mind For Mary, the abnormality of the mind would be her post traumatic stress disorder, which is recognised as a medical condition. The jury would decide if he is suffering for this by looking at medical evidence. Explanation Marys PTSD would have be part of the explanation of why she acted in the way she did, and killed Peter. PTSD can be triggered by sights, sounds and feelings they experience, for example being shouted at by Peter. This trigger can cause an intense emotion and physical reaction, stabbing Peter with a bayonet. Impaired mental ability Mary would also have to show that her PTSD affected her own mental ability to form a rational judgement or to exercise her self-control. The Jury would then use medical evidence to decide if this is the case. Provocation Case Mary and Peter Evidence She would need to have evidence of Peter insulting her and telling her that he hated her to be able to use it as part of her defence. She would be able to argue that he provoked her into stabbing him. Loss of Control This could also form part of Marys defence. This is because she had been soldier, and was suffering from post-traumatic stress disorder. She could say that it affected her judgement her self-control of her emotions, and that is why she acted in the way that she did. Reasonable man defence The jury would need to decide if a reasonable person would act in the same way as Mary did in that situation. This is difficult to use because it is very objective, and based on the juries opinion. Provocation Case Sarah and Michael. Evidence Sarah would have been provoked by Michael smirking because about sexually abusing young boys. This could also be affected because she is a mother. She would need to have evidence of him doing this. Michaels actions would still be considered provocation, even though Sarah had initiated it by confronting him about the sexual abuse. Loss of Control Sarah could say that the fact that he smirked at the accusation of sexual abuse provoked her, and this caused her to lose her self-control. That would explain why she acted in the way she did. Reasonable man defence The jury would need to decide if a reasonable person would act in the same way as Sarah. This is difficult to use because it is very objective, and based on the juries opinion. Diminished Responsibility wouldnt apply to the case of Sarah and her Neighbour as there is no evidence that she had an abnormality of the mind. The fact that she   was drunk would not be able to be used as evidence, because this defence requires it to be a history of alcoholism.

Americas First Serial Killer :: essays research papers

In a later time, Minnie R. Williams would have been called a â€Å"Dallas girl.† She grew up in Big D, received a quality education at Boston’s Conservatory of Elocution and she had money. The money came following the death of the kindly rich uncle who had taken her in as an orphan. Minnie inherited his estate, including some real estate in Fort Worth appraised at nearly $50,000. After her out-of-state schooling, Minnie lived in Dallas with her sister Nannie (who had been raised by another uncle) before deciding to return to Boston for additional studies. While there, she met a good looking fellow named Harry Gordon. In addition to being handsome, he was smart. And, like Minnie, he had money – or at least said he did. Smitten from the start, Minnie soon signed letters to her sister as Mrs. Harry Gordon. The newlyweds moved to Chicago in March 1893. Later that spring, Minnie wrote Nannie and asked her to come to Chicago to see the world’s fair then under way. The sister arrived in June. In early July, Nannie wrote her aunt that she, Minnie and her new brother-in-law planned to visit Europe. That letter, written July 4, 1893, was the last time anyone ever heard from either of the two sisters. Later that summer, their relatives engaged the famous Pinkerton Detective Agency to find the two young women. Meanwhile, an affable Midwesterner got off the train in Fort Worth to handle a little financial business. At the Tarrant County Clerk’s office he filed a deed signed over to him by his wife, Minnie Williams Gordon, preparatory to making improvements to the property. Gordon soon hired a contractor to build an expensive three-story stone and frame building on the lot. With construction still under way, Gordon borrowed $20,000, using his real estate and the planned improvements as collateral. But Gordon seemed to have trouble making the loan payments. Before long, he borrowed something else – someone’s horse – and â€Å"consolidated† his debt by riding that horse out of state with most of the bank’s $20,000 in his pocket. The Pinkerton men on the case succeeded in unraveling it, tracing Gordon to Boston. When Bean Town police arrested him on Nov. 17, 1894, he confessed to scamming folks in Texas. But he did not confess all at first. When he lived in Chicago, he said, â€Å"I fell in with a typewriter girl [Minnie] and furnished a house on the outskirts of the city, where we lived together.

Red Badge Of Courage Paper -- essays research papers

During the Civil War, a Union regiment rests along a riverbank, where they have been camped for weeks. The tall soldier Jim Conklin spreads a rumor that the army will soon march. Jim is a man who is very sure of himself and his own opinions. Henry Fleming, a recent recruit with the 304th regiment, worries about his courage, thinking that if he were ever to see a battle, he might run. Henry joined the army because he was drawn to the glory of military conflict, but since he joined, all the army has done is wait.   Ã‚  Ã‚  Ã‚  Ã‚  At last the regiment is given the orders to march, and the soldiers spend several wearying days traveling on foot. Eventually they near a battlefield, and they begin to hear the distant roar of battle. After taking their positions, they were charged by the enemy; Henry, boxed in by his fellow soldiers, realizes that he could not run even if he wanted to. He fires mechanically, feeling like a gear in a huge machine. The blue regiment defeated the gray soldiers, and then men congratulate one another. Henry wakes from a brief nap to find that the men are being charged again. This time, terror overtakes him, and he leaps up and flees from the line. As he dodges through the landscape, he tells himself that he did the right ting to flee, that his regiment could not have won, and the men who remained to fight were fools. But he passes a general on horseback, and overhears the commander saying that the men have held back the enemy charge. Feelin...

Kamote tops Essay

Sweet Potato Leaves (talbos ng kamote) Sweet potato tops (kamote tops or talbos ng kamote) are the shoots and leaves of the sweet potato plant that contains protein, lipids, carbohydrates, calcium, iron, phosphorus and vitamins A and C. It is excellent source of anti-oxidative compounds, mainly polyphenolics, which may protect the human body from oxidative stress that is associated with many diseases including cancer and cardiovascular diseases. It is relatively low in calories and has no fat and has the highest content of total polyphenolics among other commercial vegetables studied. Studies also indicate that it can also be used as a cure for diabetes particularly the purplish form. Research also shows that the nutritional content of kamote tops or sweet potato greens can counter attack the downfall of the platelet count of anyone hit by dengue fever by drinking its juice. It boosts the immune system of our body helping our body’s defenses against this sickness. The juice is also great for people with hemorrhaging problems like internal cervical bleeding and intestinal bleeding. It is prepared by boiling the leaves adding a little salt, sugar, or kalamansi to make it easier to drink. Since it is easy to grow, you can find these leaves anywhere and in the Philippines. In less than a month from planting, you can start harvesting the tops and leaves thus it is usually organic and safe making it more healthy and nutritious. While it is usually added as vegetable to dishes like sinigang, it can be eaten fresh in salads or with shrimp paste or fish sauce. It can be cooked in vinegar and soy sauce and served with fried fish (Adobong talbos ng kamote). It can also be sautà ©ed in place of spinach adding them to stir-fries or a replacement to another well known green in the Philippines called Kangkong.

Reasons Why I Want to be an RN Essay

There are many reasons why I would like to be an RN, and there are many reasons of why I should become an RN, ever since I was a child I have had the idea of being a nurse implanted in my head. Then as time went by other ideas came into play, like being a teacher or being a police officer, but being a nurse always stayed in my mind as a great career for me. I got this idea of being a nurse from my mother when I was a little kid, see my mom is a nurse, actually she is now the director of nursing at Bakersfield Heart. But when I was a kid, she went from floor nurse, to charge nurse, to director of her floor, to house supervisor, to assistant director, and now director. I can easily see her retiring as the CEO of this hospital. Now I do not know if I would want to do all of that, but I definitely see myself going from floor nurse to charge nurse, and maybe the director of the floor. The upper management just does not sound as fun to me. So originally I did not pursue the idea of nursing, I actually went into college thinking I was going to be a local high school teacher and wrestling coach, so I went and got my degree in Art Education, but while I was in college, I started working as a transport orderly at San Jose Medical Center before it closed, then went to Regional Medical Center of San Jose, where I started working as an Emergency Room clerk, and moved up to an Emergency Room Technician and loved the hands on with the patients. This is where I decided that teaching may wait till I retire from nursing, and nursing will become my career. Soon as I started working side by side with our nurses in the ER I saw that this is what I should be doing, and this is what I want to be doing, so I started working on the prerequisites right then and there. I loved the compassion the nurses had for their patients, and to see the smiles that would come from these patients when they felt better. There was something about being bedside that just pushed me to want to be more. Being a tech was and is amazing, but I want more, I want to be running the code, not just  a part of the code, when a trauma victim comes in, or a coded patient comes in. I love the feeling of bring back a patient in a code, even though I know right now by doing just CPR I am the one that is moving the medication through the body, I want to be the one that is pushing epinephrine, to bring that patient back. In this world today, I feel like there are less and less people that are actually trying to improve today’s society, and are looking out only for themselves, or their family. And I do not want to be one of those people. I want to be helping the community whether it is to clean up a homeless man, or save the life of a kid that was shot, you never know what that person can do for the rest of their lives to improve someone else’s life. I know that not all nursing is emergency nursing, but it all has its parts to the community and helping others. And I am ok with that, but I truly see myself in emergency nursing. If you notice that all of my jobs I was looking at while I was growing up, nursing, teaching, police officer, they are all helping professions, they are all improving the world around us, and ultimately that is what I really want to do, is improve the community, and improve the well being of the people around me. And I believe nursing is the right form of profession for me to do that.